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Faculty of Medicine College Hospital Orthopedics department |
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Assistant Professor |
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Related SDGs |
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KUROGI Syuji
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Papers 【 display / non-display 】
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脊椎手術の術中・術後の止血に難渋し発覚した血小板機能異常症の1例
肥後 聖, 比嘉 聖, 濱中 秀昭, 黒木 修司, 永井 琢哉, 日髙 三貴, 高橋 巧, 帖佐 悦男
整形外科と災害外科 73 ( 3 ) 415 - 420 2024.9
Authorship：Lead author, Last author, Corresponding author Language：Japanese Publishing type：Research paper (scientific journal) Publisher：西日本整形・災害外科学会
【はじめに】凝固能の評価のために手術前には凝固能検査を行うことが一般的である．今回，脊椎手術の術中・術後の止血に難渋し発覚した血小板機能異常症の1例を経験したため報告する．【症例】74歳男性，間欠跛行を主訴に当院受診され，MRI検査にてL3/4/5に変性すべり症に伴う腰部脊柱管狭窄症を認めた．腰椎固定術を施行し，術中に軟部組織からの出血が多く止血に難渋し，また術後ドレーンや創部からの多量の出血を認めた．過去にも当院で腰部脊柱管狭窄症に対して椎弓切除術施行した際に術後血腫を認め血腫除去術を施行していた．父親も痔核術後に大量出血で逝去した家族歴があるため遺伝性血液凝固異常があると考えられた．精査の結果，遺伝性の血小板機能異常症の診断となった．【考察】脊椎手術の術中・術後の止血に難渋し発覚した血小板機能異常症の1例を経験した．家族歴があり術中術後の止血に難渋する場合は血小板機能異常症を考慮する必要がある．
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Ureter position and risk of ureteral injury during lateral lumbar interbody fusion Reviewed
Hamanaka H., Tajima T., Kurogi S., Higa K., Nagai T., Kuroki T., Takamori H., Mukai S., Kamoto T., Chosa E.
Journal of Orthopaedic Science 29 ( 3 ) 775 - 780 2024.5
Authorship：Lead author, Last author, Corresponding author Language：English Publishing type：Research paper (scientific journal) Publisher：Journal of Orthopaedic Science
Background: Ureteral injury during lateral lumbar interbody fusion (LLIF) is uncommon. However, it is a serious complication that may require additional surgery should it occur. The objective of this study was to evaluate whether there was any change in the position of the left ureter between preoperative biphasic contrast-enhanced CT scanned in the supine position and intraoperative scanning in the right lateral decubitus position after stent placement, to assess the risk of ureteral injury in the actual surgical position. Methods: The position of the left ureter scanned with the O-arm navigation system with the patient in the right lateral decubitus position and its position on preoperative biphasic contrast-enhanced CT images scanned with the patient in the supine position were investigated comparing their positions at the L2/3, L3/4, and L4/5 levels. Results: The ureter was located along the interbody cage insertion trajectory in 25 of 44 disc levels (56.8%) in the supine position, but in only 4 (9.5%) in the lateral decubitus position. The proportion of patients in whom the left ureter was located lateral to the vertebral body (along the LLIF cage insertion trajectory) at each level was 80% in the supine position and 15.4% in the lateral decubitus position at the L2/3 level, 53.3% in the supine position and 6.7% in the lateral decubitus position at the L3/4 level, and 33.3% in the supine position and 6.7% in the lateral decubitus position at the L4/5 level. Conclusion: The proportion of patients in whom the left ureter was located on the lateral surface of the vertebral body when the patient was in the actual surgical position (lateral decubitus position) was 15.4% at the L2/3 level, 6.7% at the L3/4 level, and 6.7% at the L4/5 level, suggesting that caution is required during LLIF surgery.
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Risk of Skull Perforation with Halo Vest Skull Pins
Hamanaka Hideaki, Tajima Takuya, Kurogi Syuji, Higa Kiyoshi, Nagai Takuya, Takahashi Takumi, Matsumoto Takayuki, Chosa Etsuo
Spine Surgery and Related Research 8 ( 5 ) 480 - 484 2024
Authorship：Lead author, Last author, Corresponding author Language：English Publishing type：Research paper (scientific journal) Publisher：The Japanese Society for Spine Surgery and Related Research
Introduction: A halo vest is an immobilization device widely used to stabilize the cervical spine. Pain and infection at the skull pin insertion site are common complications, but skull perforation is rare, and most published studies are case reports. This study aimed to identify risk factors for skull perforation by comparing patients who did and did not develop perforation.Methods: Overall thickness and the thicknesses of the internal and external laminae of the skull at the skull pin insertion sites were measured on cranial computed tomography scans of 66 patients fitted with a halo vest. The results were compared between patients who did and did not develop perforation.Results: Four patients developed perforations. All patients with perforation were older women, and their external and internal laminae were significantly thinner than those of patients who did not develop perforation.Conclusions: The reported causes of skull pin perforation include infection around the pin, osteoporosis, and an enlarged frontal sinus. However, most patients with perforation in the present study were older women, and the cause was the thinning of the external and external laminae.
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Tmem161a regulates bone formation and bone strength through the P38 MAPK pathway
Nagai Takuya, Sekimoto Tomohisa, Kurogi Syuji, Ohta Tomomi, Miyazaki Shihoko, Yamaguchi Yoichiro, Tajima Takuya, Chosa Etsuo, Imasaka Mai, Yoshinobu Kumiko, Araki Kimi, Araki Masatake, Choijookhuu Narantsog, Sato Katsuaki, Hishikawa Yoshitaka, Funamoto Taro
Scientific Reports 13 ( 1 ) 14639 2023.12
Authorship：Lead author, Last author, Corresponding author Language：English Publishing type：Research paper (scientific journal)
Bone remodeling is an extraordinarily complex process involving a variety of factors, such as genetic, metabolic, and environmental components. Although genetic factors play a particularly important role, many have not been identified. In this study, we investigated the role of transmembrane 161a (Tmem161a) in bone structure and function using wild-type (WT) and Tmem161a-depleted (Tmem161aGT/GT) mice. Mice femurs were examined by histological, morphological, and bone strength analyses. Osteoblast differentiation and mineral deposition were examined in Tmem161a-overexpressed, -knockdown and -knockout MC3T3-e1 cells. In WT mice, Tmem161a was expressed in osteoblasts of femurs; however, it was depleted in Tmem161aGT/GT mice. Cortical bone mineral density, thickness, and bone strength were significantly increased in Tmem161aGT/GT mice femurs. In MC3T3-e1 cells, decreased expression of alkaline phosphatase (ALP) and Osterix were found in Tmem161a overexpression, and these findings were reversed in Tmem161a-knockdown or -knockout cells. Microarray and western blot analyses revealed upregulation of the P38 MAPK pathway in Tmem161a-knockout cells, which referred as stress-activated protein kinases. ALP and flow cytometry analyses revealed that Tmem161a-knockout cells were resistant to oxidative stress. In summary, Tmem161a is an important regulator of P38 MAPK signaling, and depletion of Tmem161a induces thicker and stronger bones in mice.
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頸部傍脊椎領域原発悪性リンパ腫に対し椎弓形成術を施工した小児の１例 Reviewed
飯田暁人、濱中秀昭、黒木修司、比嘉聖、永井琢哉、黒木智文、日髙三貴、帖佐悦男
整形外科と災害外科 72 ( 4 ) 735 - 738 2023.9
Language：Japanese Publishing type：Research paper (bulletin of university, research institution) Publisher：西日本整形・災害外科学会機関誌
MISC 【 display / non-display 】
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脊椎 Scapulohumeral reflex Invited
黒木修司、帖佐悦男
関節外科 37 ( 9 ) 114 - 115 2018.9
Language：Japanese Publishing type：Article, review, commentary, editorial, etc. (trade magazine, newspaper, online media) Publisher：メディカルビュー
Presentations 【 display / non-display 】
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Col2A1異常に関連した胸椎後側弯症の1例
永井琢哉、黒木修司、比嘉聖、高橋巧、松本尊行、帖佐悦男
第39回九州小児整形外科集談会 2024.1.13
Event date： 2024.1.13
Presentation type：Oral presentation (general)
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腰椎手術中・術後に止血困難で治療に難渋した血小板機能異常症の経験
肥後聖、比嘉聖、濱中秀昭、黒木修司、永井琢哉、日髙三貴、高橋巧、帖佐悦男
第146回西日本整形・災害外科学会学術集会 2023.11.3
Event date： 2023.11.3 - 2023.11.4
Presentation type：Oral presentation (general)
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Tmem161aはP38 MAPK経路を介し骨形成と骨強度を制御する重要な因子である
永井琢哉、関本朝久、山口洋一朗、黒木修司、舩元太郎、田島卓也、今坂舞、吉信公美子、荒木喜美、荒木正健、帖佐悦男
第38回日本整形外科学会基礎学術集会 2023.10.20
Event date： 2023.10.19 - 2023.10.20
Presentation type：Oral presentation (general)
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骨粗鬆症はハローベストのスカルピン頭蓋骨穿破のリスクとなる
濱中秀昭、黒木修司、比嘉聖、永井琢哉、黒木智文、日髙三貴、帖佐悦男
第25回日本骨粗鬆症学会 2023.9.29
Event date： 2023.9.29 - 2023.10.1
Presentation type：Oral presentation (general)
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胸腰椎破裂骨折に対する経皮的椎弓根スクリューを用いた後方固定術における椎間関節の骨癒合の検討
黒木智文、濱中秀昭、黒木修司、比嘉聖、永井琢哉、日高三貴、帖佐悦男
第145回西日本整形・災害外科学会学術集会 2023.6.3
Event date： 2023.6.3 - 2023.6.4
Presentation type：Oral presentation (general)